Math1基因,Math1英语短句,例句大全

Math1基因,Math1
1)Math1Math1基因
1.At this stage,Math1 gene transfer should be effective and it is possible that inner ear morphology and function can restore.针对内耳基因治疗时间窗问题,我们根据听觉损伤后不同的病理状态,提出听觉损伤后毛细胞再生和基因治疗的基本策略:(1)毛细胞纤毛损伤阶段,是基因治疗的最好时机,通过完全修复或纤毛再生达到功能的完全或部分恢复;(2)内耳毛细胞虽有损伤但没有坏死,支持细胞和神经纤维基本正常,所以有恢复形态和功能的机会,这个阶段导入Math1基因应该有效,是基因治疗的最关键时机;(3)毛细胞严重损伤但支持细胞尚存,是毛细胞再生的抢救阶段,而且还可以争取在Corti器细胞构架没有塌陷之前进行干细胞导入,所以这个阶段内细胞移植可能有效地实现听力恢复;(4)Corti器完全失去构架,仅仅残留上皮层或瘢痕化,基因导入完全无效,即使干细胞导入也会面临困难,如何重塑Corti器构架是巨大挑战。

英文短句/例句

1.The surgical approach of Math1 gene transfer into cochlea in the guinea pigMath1基因内耳导入径路的探索研究
2.Evaluation for the best way of Math1 gene transfering into vestibule in adult ratMath1基因导入成年大鼠前庭有效途径的探索
3.Effects of Lipofectemine Mediated bFGF-Math1 Gene Transfer to UEC-4 Cell on Reduction and Differentiation;脂质体介导的bFGF-Math1基因在UEC-4细胞中的表达及诱导分化作用
4.Study on Cochlea Hair Cell Regeneration in Presbycusis Mice by Math1 Gene Inner Ear Transfection老年性聋小鼠Math1基因导入诱导耳蜗毛细胞再生的研究
5.The Study on the Vestibular Hair Cell Regeneration of Adult Rat Induced by Math1 Gene Transfer in VivoMath1基因诱导成年大鼠前庭毛细胞再生的在体实验研究
6.Hearing improvement of NIHL guinea pig by overexpressed Math1 gene in the cochleaMath1基因内耳导入后噪声性聋豚鼠听功能改变观察
7.Study on transfection efficiency and cytotoxicity of 293T cells with different vectors containing Math1-EGFP gene不同类型载体携带Math1-EGFP基因对293T细胞转染效率及细胞毒性的研究
8.The Expression of Math1 Induced by Sonic Hedgehog in the Inner Ear刺猬蛋白促进内耳Math1表达的实验研究
9.extinguisher loci消失基因座，绝灭基因座
10.Of, relating to, produced by, or being genes or a gene.基因的基因的，与基因有关的，基因产生的
11.Genes are described as DOMINANT or RECESSIVE.基因分为显性基因和隐性基因。
12.hypomorphic allele次等位因素对偶基因
13.Studies on Polymorphism of Prolactin Receptor Gene and Follicle-Stimulating Hormone Beta Subunit Gene in Swine;猪PRLR基因和FSHβ亚基基因多态性研究
14.Research on Hte Microarray Based Gene Mining Algorithm;基于基因表达谱的基因挖掘算法研究
15.Human Genome Project,Post-genome Research and Gene Medicine --New Century of Diagnosis,Pharmaceutics and Therapy with Gene Technique;人类基因组计划、后基因组研究与基因医学——基因诊断、基因制药与基因治疗的新世纪
16.Genetic material produced by gene-splicing.重组基因材料基因分割而产生的基因材料
17.Gene distribution was out of balance. Dominant genes were more than recessive ones.基因的分布是不对称的,显性基因多于隐性基因。
18.Association of apoE Gene, A2M Gene and ACE Gene Polymorphism with Alzheimer s Disease;apoE基因、A2M基因、ACE基因与汉人Alzheimer病的相关性研究

相关短句/例句

gene[英][d?i:n][美][d?in]基因
1.Study of the Preparation and the Characteristics of pRc/CMV-BChE Gene-chitosan nanoparticles ①;丁酰胆碱酯酶基因-壳聚糖纳米粒初步研究
2.Correlation between traditional Chinese medicine syndromes in primary immunoglobulin A nephropathy and A267G in 5'-untranslated region within exonal of megsin gene;Megsin基因E1-5’UTR区A267G与免疫球蛋白A型肾病阴虚证的相关性
3.Study on gene chip of leiomyoma of uterus;子宫肌瘤基因芯片的研究
3)Genes基因
1.Study on Apoptosis of Human Leukemia Cells and Its Related Genes Regulation Induced by ~(235) U;浓缩铀诱发细胞凋亡的形态及基因调控
2.Screening of lymphatic metastasis-associated genes in esophageal squamous cell carcinoma;食管癌淋巴结转移相关基因筛选的研究
3.Study on the Genotyping of Aminoglycoside Modifying Enzymes Genes from Pan-drug Resistant Acinetobacter Baumannii.;泛耐药鲍曼不动杆菌氨基糖苷类修饰酶基因研究
4)Polymorphism[英][,p?li'm?:fiz?m][美][,pɑl?'m?rf?zm?]基因
1.The Study on Apolipoprotein E Gene Polymorphism Characteristics of Cerebral Infarction and Intracerebral Hemorrhage;脑梗死与脑出血apoE-基因多态性特点研究
2.The Relations of Gene Polymorphisms of eNOS and FⅦ with Coronary Heart Disease in Henan Han Population;河南汉族人群一氧化氮合酶和凝血因子Ⅶ基因多态性与冠心病相关性分析
3.Methods:Polymorphism of the 677th site C/T of MTHFR gene and the 66th site A/G of MTRR gene were detected by polymerase chain reaction-restrction fragment length polymorphism in disease group(n=64) and normal controls(n=104).目的:研究同型半胱氨酸相关酶中亚甲基四氢叶酸还原酶(MTHFR)及蛋氨酸合成酶还原酶(MTRR)基因的多态性与先天神经管缺陷的关系。
5)AS geneAS基因
6)Genetic[英][d??'net?k][美][d??'n?t?k]基因
1.Antigenic and genetic characterization of A/Shenzhen/1/99(H3N2) virus;A/深圳/1/99(H3N2)病毒抗原性及基因特性研究
2.Relationship between the genetic polymorphisms of VDR and susceptibility to pulmonary tuberculosis among the Chinese Han population维生素D受体基因多态性与汉族人肺结核发病的关系
3.MTHER genetic C677T polymorphisms were determined by PCR-RFLP.N5, 10 亚甲基四氢叶酸还原酶(MTHFR)是参与甲硫氨酸-叶酸代谢的关键酶,其基因677位点C→T错义突变可造成此酶活性降低,导致高同型半胱氨酸血症,现已证明高同型半胱氨酸是诱发胎儿出生缺陷和心血管疾病的一个独立危险因素。