1)signal mechanisms细胞内信号机制
1.These data suggest that CCL2 might play a major role in the neuropathic pain, but the underlying signal mechanisms of CCL2 have not been elucidated yet.为进一步研究CCL2对痛觉的效应及其细胞内信号机制,本课题在慢性压迫背根神经节的模型上,应用western blot和免疫组织化学染色技术明确CCD大鼠背根神经节(DRG)细胞信号分子蛋白的表达与分布;应用行为学测试方法观察CCL2对CCD大鼠痛觉行为的作用及其可能机制;应用电生理技术观察了CCL2对CCD大鼠DRG神经元兴奋性及钠电流的作用及其可能的细胞内信号途径。
英文短句/例句
1.Signal Mechanisms Mediated the Roles of Chemokine CCL2 in CCD-induced Neuronal Hyper-excitability and Mechanical Hyperalgesia趋化因子CCL2增强痛觉效应的细胞内信号机制
2.Regulatory Effect of Caveolin-1 on Endothelial Cell Proliferation;高表达Caveolin-1抑制内皮细胞增殖的信号传递机制研究
3.STUDY OF SIGNAL TRANSDUCTION MECHANISMS OF PBMC AFTER SEVERELY BURNS严重烧伤致单个核细胞内信号转导机制变化的实验研究
4.Effects of Endogenous Nitric Oxide on Apoptosis of Gastric Cancer Cells and Its Singal Transduction Pathways;内源性一氧化氮对胃癌细胞凋亡的影响及信号转导机制研究
5.The Mechanism of Signal Transduction in Human Monocyte Stimulated by LPS and the Effect of Chinese-drugs on It;内毒素激活人单核细胞的信号传导机制及中药对其影响
6.Effects of Astragalus Membranaceus Injection on Endothelial Progenitor Cells from Human Peripheral Blood and Its p38 MAPK Signaling Pathways Mechanisms;黄芪注射液对人外周血内皮祖细胞的影响及p38MAPK信号传导机制研究
7.Mechanism of Ursolic Acid Inhabiting the Proliferation of Gliocytoma and Vascular Endothelialcell by ERK Extracelluar Signaltransduction Pathway熊果酸经ERK信号转导通路抑制星型胶质细胞瘤及血管内皮细胞增殖的分子机制
8.Studies on the Role and Its Intracellular Signal Transduction Mechanism of Postasphyxial-serum of Neonate in Inducing Injury of Human Renal Tubular Cells(HK-2);新生儿窒息后血清诱导HK-2细胞损伤的作用及其细胞内信号传导机制的研究
9.Dapper1 is a Nucleocytoplasmic Shuttling Protein That Negatively Modulates Wnt Signaling in the NucleusDapper1是核质穿梭蛋白并在细胞核内抑制Wnt信号
10.Silencing effect of RNA interference on suppressor of cytokine signaling-1 in human umbilical vein endothelial cellsRNAi对细胞因子信号转导抑制因子1在内皮细胞中表达的沉默作用
11.The Signal Mechanism Underlying Basic Fibroblast Growth Factor-mediated Antiapoptosis in Ovarian Cancer CAOV3 Cells;碱性成纤维细胞生长因子抑制卵巢癌CAOV3细胞凋亡的信号转导机制
12.Effect of Anti-apoptosis Drugs on Signal Transduction of Lens Epithelial Cell Damaged by Oxidative Stress;细胞凋亡抑制剂防护晶状体上皮细胞氧化损伤的信号转导机制
13.Role and Signalling of Endoplasmic Reticulum Stress in Endogenuous Cardiomyocyte of Hypoxic Preconditioning and Hypoxic Postconditioning;内质网应激在内源性心肌细胞保护中的作用及其信号转导机制研究
14.The Study of Multipotent Adult Progenitor Cells Endothelial Differentiation & the Effect of High Glucose on Its Differentiation and Correlated Signaling Pathway;多潜能成体干细胞内皮分化和高糖对内皮分化的影响及相关信号通路机制探讨
15.Study on the Mechanism of Deltamethrin-induced Neurocytes Apoptotic Signal Transduction;溴氰菊酯神经细胞凋亡信号转导机制的研究
16.Mechanism of Action of SAP and PIX in Immune Cells;SAP与PIX在免疫细胞信号传导中作用机制的研究
17.The Study of Signal Transduction Mechanism of Histamine H_3 Receptor on AtT-20 Cell;组胺H_3受体在AtT-20细胞中信号转导机制的研究
18.The Study on the Mechanotransduction Mechanism of Mandibular Condylar Chondrocyte;下颌髁突软骨细胞力学信号转导机制的研究
相关短句/例句
cell signaling mechanisms细胞内的信号转导机制
3)intracellular signal transduction细胞内信号传导
4)cell signal transduction细胞内信号转导
5)cell signaling细胞信号
6)extracellular signal胞内信号
延伸阅读
脉心导敏 ,脉导敏,英他佐明,麦新导明药物名称:吗多明英文名:Molsidomine别名: 吗导敏;吗多明;吗斯酮胺;脉心导敏 ,脉导敏,英他佐明,麦新导明外文名:Molsidomine 特点: 脉导敏是一种属于亚胺类化合物,其作用与硝酸盐相似。舌下含服1毫克,2~4分钟起作用,可阻止心绞痛发作。口服一次可持续6~7小时,剂量每天3次,每次2毫克。副作用包括头痛、头胀、脸部发热感等,但不多见。 适应症: 防治心绞痛的发作。 用量用法: 口服:1次1~2mg,1日2~3次,发作时舌下含服:1次2mg。喷雾吸入:每次揿吸1~2次(相当于本品0.2~0.4mg),每日次数酌定。 注意事项: 1.一般不良反应可有头痛、面部潮红、眩晕等,停药后可自行消失。 2.低血压、青光眼病人忌用。 规格: 片剂:每片1mg、2mg。 气雾剂:每瓶含42mg(可揿吸200次左右)。 类别:非选择性钙通道阻滞剂||防治心绞痛药