狼疮凝固抑制物,Lupus coagulation inhibitor
1)Lupus coagulation inhibitor狼疮凝固抑制物
2)Lupus anticoagulant狼疮抗凝物
3)Lupus anticoagulant狼疮抗凝物质
1.Objective To measure lupus anticoagulant(LA),activated protein C-resistant(APC-R) and indicate of hypercoagulation states in patients with Cerebral Infarct and to research their relation and set up a serial programs to laboratory diagnosis,monitoring treatment and mechanism of Cerebral Infarct.目的 检测脑梗塞患者体内的高凝状态指标及狼疮抗凝物质 ( lupus anticoagulant,LA)、活化蛋白 C抵抗 ( activated protein C- resistant,APC- R) ,探讨 LA、APC- R与脑梗塞患者高凝状态的关系 ,为脑梗塞的实验诊断、治疗监测及发生机制的研究提供依据。
2.There was a medium to strong positive lupus anticoagulant(LA).在1个月内发生双上臂伸侧、双肋胁部及两侧腹部下方多处皮肤梗死,同时伴有脾内多发性栓塞、不完全性肠梗阻及自身免疫性血小板减少(ITP),实验室检查显示狼疮抗凝物质(LA)中至强阳性。
4)Iupus coagulation inhibitor狼疮样抗凝物质
英文短句/例句

1.Effects of lupus anticoagulant on blood coagulation in patients with chronic kidney disease狼疮样抗凝物质对慢性肾脏疾病患者凝血功能的影响
2.Thromboembolism and anticoagulant therapy in patients with lupus nephritis狼疮性肾炎合并血栓栓塞与抗凝治疗
3.The relationship of prethrombotic state with antiphospholipid antibody and anticoagulant protein system in systemic lupus erythematosus patients抗磷脂抗体和抗凝蛋白系统与系统性红斑狼疮血栓前状态的关系
4.Effects of leflunomide on expression of BMP-7,TGF-β1 in renal tissue of murine chronic graft-versus-host disease lupus nephritis来氟米特对慢性移植物抗宿主病狼疮样小鼠肾组织BMP-7、TGF-β1表达的影响
5.lupus vulgaris【医】红斑样寻常狼疮
6.Effects of Qdjn on Serum IL-10 and Anti ds-DNA in SLE-LIKE Mouse;祛毒胶囊对狼疮样小鼠外周血IL-10和抗ds-DNA抗体的影响
7.Conlusion The mouse model of chronic GVHD, which is alike to human lupus nephritis, can be used as a good model of lupus nephritis.结论 慢性移植物抗宿主模型类似人类狼疮性肾炎 ,是良好的狼疮肾炎模型。
8.The Research of Lupus-like Mouse Model Estabilised by Sm Mimotopes and Interfered by CRP;利用Sm抗原表位构建狼疮样鼠模型及C反应蛋白干预的研究
9.Preliminary Study of Sm Antigen Mimic Epitope Peptide-induced Immune Tolerance Therapies Against Systemic Lupus Erythematosus Like Mice;Sm抗原表位肽诱导狼疮样鼠免疫耐受的初步实验研究
10.Conclusion: LALP has anticoagulant effect in vivo, and the effe ctive mechanism may interfere with phosphatide.结论:类狼疮抗凝蛋白具有体内抗凝血作用,作用机制可能与其干扰磷脂有关。
11.The Relationship of Venous Thromboembolism with Antiphospholipid Antibody and Anticoagulant Protein System in Systemic Lupus Erythematosus Patients抗磷脂抗体和抗凝蛋白系统与系统性红斑狼疮静脉血栓形成的关系
12.The Effects on BALB/c Mice Lupus-like Symptoms Induced by the Mixture of Paraphenylenediamine and H_2O_2对苯二胺与双氧水混合物诱导小鼠狼疮样改变的作用
13.The effect of the mixture of paraphenylenediamine and H_2O_2 in inducing BALB/c mice to develop lupus-like symptoms对苯二胺与H_2O_2混合物诱导小鼠狼疮样改变的作用
14.The role of auto-antibodies related to disease activity of systemic lupus erythematosus and associated renal renal involvement抗自身抗体与系统性红斑狼疮疾病活动性及狼疮肾炎关系的探讨
15.Abnormality of Mesenchymal Stem Cells (MSCs) in Patients with Lupus and Experimental Study of Umbilical Cord MSCs Transplantation in Lupus Mice红斑狼疮间质干细胞(MSCs)异常及脐带MSCs移植治疗狼疮鼠的实验研究
16.Induction of Systemic Lupus Erythematosus Syndrome in Mice and Anti-inflammatory and Immunosuppressive Action of Tripterine;小鼠狼疮样综合征模型的建立以及雷公藤红素抗炎免疫抑制作用的研究
17.The Study of Clinical Sense of Antinucleosome Antibodies (AnuA) in Systemic Lupus Erythematosus(SLE);抗核小体抗体测定在系统性红斑狼疮中的意义
18.New Insights into the Role of IgG Anti-dsDNA Antibody and Calreticulin in Lupus Nephritis;IgG型抗dsDNA抗体和钙网蛋白在狼疮肾炎中的作用
相关短句/例句

Lupus anticoagulant狼疮抗凝物
3)Lupus anticoagulant狼疮抗凝物质
1.Objective To measure lupus anticoagulant(LA),activated protein C-resistant(APC-R) and indicate of hypercoagulation states in patients with Cerebral Infarct and to research their relation and set up a serial programs to laboratory diagnosis,monitoring treatment and mechanism of Cerebral Infarct.目的 检测脑梗塞患者体内的高凝状态指标及狼疮抗凝物质 ( lupus anticoagulant,LA)、活化蛋白 C抵抗 ( activated protein C- resistant,APC- R) ,探讨 LA、APC- R与脑梗塞患者高凝状态的关系 ,为脑梗塞的实验诊断、治疗监测及发生机制的研究提供依据。
2.There was a medium to strong positive lupus anticoagulant(LA).在1个月内发生双上臂伸侧、双肋胁部及两侧腹部下方多处皮肤梗死,同时伴有脾内多发性栓塞、不完全性肠梗阻及自身免疫性血小板减少(ITP),实验室检查显示狼疮抗凝物质(LA)中至强阳性。
4)Iupus coagulation inhibitor狼疮样抗凝物质
5)Lupus-like anticoagulants狼疮样抗凝物
6)drug-induced lupus药物性狼疮
延伸阅读

C1酯酶抑制物C1酯酶抑制物  是神经氨基糖蛋白,主要在肝内合成,血管活性肽包括纤维蛋白溶酶,激肽酶都受C1酯酶抑制物所抑制。如果缺乏,可使患病部位的血管渗透性过分增高,导致局部水肿。