线粒体通透转运孔道,mitochondrial permeability transition pore (mPTP)英语短句,例句大全

线粒体通透转运孔道,mitochondrial permeability transition pore (mPTP)
1)mitochondrial permeability transition pore (mPTP)线粒体通透转运孔道
1.The mitochondrial permeability transition pore (mPTP) is a multi-component protein at contact sites of the inner with the outer mitochondrial membrane.线粒体通透转运孔道(mitochondrial permeability transition pore,mPTP)是位于线粒体内、外膜之间的多蛋白复合体,可影响线粒体及细胞的正常生理功能。
2)mitochondrial permeability transition pore线粒体通透性转变孔道
1.Toxicity of intracellular amyloid-βpeptide and mitochondrial permeability transition pore;β-淀粉样肽的细胞内毒性与线粒体通透性转变孔道
3)permeability transition pore线粒体渗透性转运通道
4)mitochondrial permeability transition pore线粒体通透性转换孔
1.The role and mechanism of mitochondrial permeability transition pore in ischemic postconditioning;线粒体通透性转换孔在缺血后处理中的作用和机制
2.The opening state of mitochondrial permeability transition pore determines the mitochondrial membrane permeability, which is crucial to regulate apoptosis: cytochrome c is released from mitochondria to cytoplasm, activates caspases via combining Apaf-1 and ATP, initiating caspases cascade, .线粒体膜通透性转换孔的开放状态决定了线粒体膜的通透性,在调节细胞凋亡中起了非常关键的作用:细胞色素c从线粒体释放到胞浆中,与凋亡蛋白激活因子-1和ATP结合可活化caspases,启动caspases的级联反应,引起底物蛋白水解、凋亡小体形成等,诱发细胞凋亡;Bcl-2和Bax是细胞凋亡过程中两个重要的调控基因,Bcl-2的表达增强会抑制细胞凋亡,而Bax的过度表达则会促进细胞凋亡;诱导细胞凋亡时发生的线粒体Ca~(2+)摄取既诱发线粒体通透性转换孔的开放也介导线粒体发生钙诱导的钙释放,胞浆高浓度的钙能激活各种蛋白激酶而诱导细胞凋亡的发生。
3.Aim: To investigate the role of mitochondrial permeability transition pore(MPTP) in the cardioprotection by remote preconditioning(RPC).目的:观察线粒体通透性转换孔在远距预处理心肌保护中的作用。

英文短句/例句

1.Mitochondrial Permeability Transition Pore and Its Relationship with Apoptosis线粒体通透性转换孔及其与细胞凋亡的关系
2.Effects of Exercise on Mitochondrial Permeability Transition Pore and Apoptosis in Rat Skeletal Muscle;运动对大鼠骨骼肌线粒体通透性转换孔和细胞凋亡的影响
3.ROLE OF MITOCHONDRIAL PERMEABILITY TRANSITION PORE IN CARDIOPROTECTION BY REMOTE PRECONDITIONING线粒体通透性转换孔在远距预处理心肌保护中的作用
4.The effect of electroacupunture on MPTP in rats with cerebral infarction电针对脑梗塞大鼠脑皮质线粒体通透性转换孔的影响
5.Effects of Adenosine Al Receptor on the Opening of Mitochondrial Permeability Transition Pore in Hypoxic Cardiocytes and Its Mechanisms;腺苷Al受体对缺氧心肌细胞线粒体通透性转换孔开放的影响及调控机制
6.Study on the Role of Changes of Brain Mitochondrial Permeability Transition Pore in Brain Damage and Its Mechanism after Cardiopulmonary Resuscitation in Rats;心肺复苏后大鼠脑线粒体通透性转换孔改变在脑损伤中的作用及其机制研究
7.Effects of High Intensive Exercise on Myocardial Mitochondrial Membrane Permeability Transition Pore of Rats;大强度运动对大鼠心肌线粒体膜通透性转换孔状态的影响
8.Effects of Mitochondrial Permeability Transition Pore Opening on Hepatocyte Apoptosis in Development of Nonalcoholicfatty Liver Disease;线粒体通透转换孔与NAFLD肝细胞凋亡的关系
9.Effect of mitochondrial permeability transition pore in antimalarial action of artemisinin线粒体通透性转移孔与青蒿素抗疟机制研究
10.ATP-sensitive potassium channel and mitochondrial permeability transition pore involve in cardioprotection of polydatinATP敏感性钾通道和线粒体通透转换孔参与白藜芦醇苷的心肌保护作用(英文)
11.The Role of Mitochondrial Permeability Transition Pore in the Rabbit Lens Epithelial Cells Apoptosis Induced by Hydrogen Peroxide;线粒体跨膜通透性转运孔调控兔晶状体上皮细胞凋亡的研究
12.The Effect of Closing of Mitochondrial Permeability Transition Pore on Immature Rabbit Heart Protection;关闭线粒体通透性转变孔对未成熟兔缺血心肌的保护作用
13.The Roles of Mitochondria Permeability Transition in Cardiomyocytes Against Anoxia-Reoxygenation Injury;线粒体通透性转换调控对心肌细胞缺氧复氧损伤的作用研究
14.Studies on Mitochondrial Permeability Transition Related Factors during Rat Liver Regeneration;大鼠肝再生时线粒体通透性转换相关因素的研究
15.Effects of the Microtubule Damage on the Opening of Mitochondrial Permeability Transition Pore in Hypoxic Cardiomyocytes.;缺氧心肌细胞微管破坏对线粒体通透性转换的影响及其机制研究
16.Studies on the Relationship between Mitochondrial Permeability Transition and Proteins Release during Rat Liver Regeneration;大鼠肝脏再生时线粒体通透性转换与蛋白释放关系的研究
17.Studies of Mitochondrial Permeability Transition and Its Regulation during Rat Liver Regeneration;大鼠肝脏再生时线粒体膜通透性转换及其调节的研究
18.Effect of inhibiting mitochondrial permeability transition before/after reoxygenation on cardiomyocytes against hypoxia-reoxygenation injury.复氧前后抑制线粒体通透性转换对心肌细胞低氧/复氧损伤的影响

相关短句/例句

mitochondrial permeability transition pore线粒体通透性转变孔道
1.Toxicity of intracellular amyloid-βpeptide and mitochondrial permeability transition pore;β-淀粉样肽的细胞内毒性与线粒体通透性转变孔道
3)permeability transition pore线粒体渗透性转运通道
4)mitochondrial permeability transition pore线粒体通透性转换孔
1.The role and mechanism of mitochondrial permeability transition pore in ischemic postconditioning;线粒体通透性转换孔在缺血后处理中的作用和机制
2.The opening state of mitochondrial permeability transition pore determines the mitochondrial membrane permeability, which is crucial to regulate apoptosis: cytochrome c is released from mitochondria to cytoplasm, activates caspases via combining Apaf-1 and ATP, initiating caspases cascade, .线粒体膜通透性转换孔的开放状态决定了线粒体膜的通透性,在调节细胞凋亡中起了非常关键的作用:细胞色素c从线粒体释放到胞浆中,与凋亡蛋白激活因子-1和ATP结合可活化caspases,启动caspases的级联反应,引起底物蛋白水解、凋亡小体形成等,诱发细胞凋亡;Bcl-2和Bax是细胞凋亡过程中两个重要的调控基因,Bcl-2的表达增强会抑制细胞凋亡,而Bax的过度表达则会促进细胞凋亡;诱导细胞凋亡时发生的线粒体Ca~(2+)摄取既诱发线粒体通透性转换孔的开放也介导线粒体发生钙诱导的钙释放,胞浆高浓度的钙能激活各种蛋白激酶而诱导细胞凋亡的发生。
3.Aim: To investigate the role of mitochondrial permeability transition pore(MPTP) in the cardioprotection by remote preconditioning(RPC).目的:观察线粒体通透性转换孔在远距预处理心肌保护中的作用。
5)mitochondrial permeability transition pore线粒体膜通透性转换孔
1.The effect of copper on the mitochondrial permeability transition pore was significant,resulting in swelling of mitochondria to some degree which was manifested by decr.结果显示,铜对线粒体膜通透性转换孔有较显著的影响,造成线粒体不同程度的肿胀,表现为线粒体悬液在540 nm处的吸光值下降,且铜浓度越高,吸光值下降越明显,在孵育6 min后3种浓度的吸光值下降百分比差异显著(P<0。
6)Mitochondrial permeability transition pore线粒体通透转换孔
1.Mitochondrial permeability transition pore and myocardial ischemia-reperfusion injury;线粒体通透转换孔与心肌缺血-再灌注损伤
2.ATP-sensitive potassium channel and mitochondrial permeability transition pore involve in cardioprotection of polydatinATP敏感性钾通道和线粒体通透转换孔参与白藜芦醇苷的心肌保护作用(英文)
3.Objectives:The purpose of this study is with randomized controlled methods,by observing the differents among the four groups of SD rats of clean grade: cytochrome C oxidase activity and mitochondrial permeability transition pore\' s activity to explore the relations between the mitochondrial oxidative damage and osteoporosis.结果:线粒体通透转换孔30min/0minOD比值比较显示,空白对照组和尼尔雌醇组与模型组有明显差异(P<0。