1)Experimental autoimmune myasthenia gravis实验性自身免疫性重症肌无力
1.Treatment of experimental autoimmune myasthenia gravis by nasal tolerance with a recombinant fragment of acetylcholine receptor conjugated to cholera toxin B subunit;与霍乱毒素B亚单位结合的重组人AChR片断经鼻腔治疗实验性自身免疫性重症肌无力
2.Objective To investigate the effect of anti-acetylcholine receptor monoclonal antibody A7 in experimental autoimmune myasthenia gravis (EAMG).目的探讨抗乙酰胆碱受体单克隆抗体A7(抗AChRmAbA7)对实验性自身免疫性重症肌无力(EAMG)的作用。
3.Objective:To investigation the effect of anti-acetylcholine receptor monoclonal antibody A7 in experimental autoimmune myasthenia gravis (EAMG) .目的:探讨抗乙酰胆碱受体单克隆抗体A7(Anti-AChR mAb A7)对实验性自身免疫性重症肌无力(EAMG)的作用。
英文短句/例句
1.STUDY ON IMMUNE STATE OF EXPERIMENTAL AUTOIMMUNE MYASTHENIA GRAVIS MICE实验性自身免疫性重症肌无力小鼠免疫功能状态的研究
2.B cell activation in experimental autoimmune myasthenia graves treated with Tα146-162-iMDCs免疫干预实验性自身免疫性重症肌无力小鼠的B细胞活化机制
3.Effect of Nasal Tolerance with Ra97-116(V108A) on Experimental Autoimmune Myasthenia Gravis;Ra97-116（V108A）鼻粘膜耐受对实验性自身免疫性重症肌无力的影响
4.Effect and Mechanism of Bone Marrow Mesenchymal Stem Cells on Experimental Autoimmune Myasthenia Gravis;骨髓间充质干细胞对实验性自身免疫性重症肌无力的影响和机制
5.The Overview of Researchs in Animal Models of Experimental Autoimmune Myasthenis Gravis实验性自身免疫性重症肌无力动物模型研究概况
6.The Relationship of CXCL13 and Myasthenia Gravis, and the Establishment of EAMG Animal Model in MiceCXCL13与重症肌无力的相关性研究及实验性自身免疫性重症肌无力小鼠模型的建立
7.Effects of Bone Marrow Mesenchymal Stem Cells on the Clinical Manifestation and the Immune Function of Experimental Autoimmune Myasthenia Gravis骨髓间充质干细胞对实验性自身免疫性重症肌无力临床及免疫功能影响的研究
8.Effects of Yijin Prescription on Clinical Symptoms,IL-2 and Bcl-2 in Experimental Myasthenia Gravis in Mice益筋方对实验性自身免疫性重症肌无力小鼠临床症状及IL-2、Bcl-2的影响
9.Effects and Mechanism of Bone Marrow Mesenchymal Stem Cells on Th1/Th2 of Experimental Autoimmune Myasthenia Gravis骨髓间充质干细胞对实验性自身免疫性重症肌无力Th1/Th2的影响及机制
10.The Role of Nasal Immune Tolerance on the Expression of Fas and Bcl-2 in the Thymus of Experimental Autoimmune Myasthenia Gravis Rats鼻粘膜耐受对实验性自身免疫性重症肌无力大鼠胸腺Fas、BCL-2表达的影响
11.Passive Transfer of Experimental Autoimmune Myasthenia Gravis in Rabbits with IgG of Myasthenia gravis patients被动转移法致自身免疫性重症肌无力兔模型的建立
12.Blood Compatibility Study of Immunoadsorbent in Myasthenia Gravis by Whole Blood Perfusion;全血灌流重症肌无力免疫吸附剂血液相容性实验研究
13.Effects of Thymopoietin on the Immune System in Mice Model of Myasthenia Gravis;胸腺五肽对实验性重症肌无力小鼠模型免疫功能的影响
14.The Experimental Research in Immunoregulation Effect of the Jianpiqushi Prescription on the Experimental Autoimmune Myasthenia Gravis Rats健脾祛湿方对实验性重症肌无力大鼠的免疫调节作用的研究
15.As with other autoimmune diseases, people with myasthenia gravis would be expected to benefit from intravenous immunoglobulin.如其它自身免疫性疾病一样，静脉注射免疫球蛋白或许对重症肌无力有益。
16.Extracorporeal Whole Blood Immunoadsorption of Experimental Autoimmune Myasthenia Gravis;全血灌流免疫吸附治疗重症肌无力的实验研究
17.A Study of Immunotherapy and Immunomodulation Mechanism in the Treatment of Experimental Autoimmune Myasthenia Gravis by RelB-Silenced DCRelB基因沉默DC对实验性重症肌无力的治疗作用及免疫调节机制研究
18.Preparation of specific immunoadsorbent in myasthenia gravis and its dynamic absorption test on the patients' sera重症肌无力特异性免疫吸附剂的制备及对患者血清的动态吸附实验
相关短句/例句
oEAMG实验性自身免疫性眼肌型重症肌无力
1.Objective:To establish a novel model of ocular experimental autoimmune myasthenia gravis (oEAMG) in HLA-DQ8 transgenic mice and study the immunologic pathogenesis of oEAMG .目的：利用HLA—DQ8转基因小鼠建立实验性自身免疫性眼肌型重症肌无力(oEAMG)模型，并探讨oEAMG免疫学发病机制。
3)experimental autoimmune myasthenia gravis (EAMG)实验性自身免疫性重症肌无力(EAMG)
4)EAMG实验性自身免疫重症肌无力(EAMG)
5)experimental autoimmune myositis实验性自身免疫性肌炎
1.The role of CD28 in experimental autoimmune myositis and response to tripterygium wilfordii polycoside treatment;雷公藤多甙对CD28在实验性自身免疫性肌炎发病中作用的影响
2.Objective:To study the expression of tumor necrosis factor-related apoptosis inducing ligand(TRAIL)and its receptors in muscle tissues of experimental autoimmune myositis(EAM) model in rats.目的:分析肿瘤坏死因子相关凋亡诱导配体(TRAIL)及其受体在实验性自身免疫性肌炎(EAM)动物模型骨骼肌组织中的表达情况,研究TRAIL系统在特发性炎性肌病(IIM)发病机制中的作用和意义。
6)Experimental Autoimmune Myasthenia Gravis实验性重症肌无力
1.A Study of Immunotherapy and Immunomodulation Mechanism in the Treatment of Experimental Autoimmune Myasthenia Gravis by RelB-Silenced DCRelB基因沉默DC对实验性重症肌无力的治疗作用及免疫调节机制研究
延伸阅读
重症肌无力重症肌无力myastheniagravis以骨骼肌神经肌肉接头处传递功能障碍为主的疾病。女性多见。表现为受累骨胳肌不能耐受疲劳而出现肌无力，症状晨轻暮重，休息后可以减轻，应用抗胆碱酯酶药物后症状可以迅速缓解。发病机理为体内产生自身抗体——乙酰胆碱受体(AChR）抗体，从而破坏了突触后膜上的AChR而使传递功能发生障碍。最常受累的肌肉为眼外肌，其次为四肢骨胳肌、咽喉部肌肉。如侵犯呼吸肌出现呼吸肌瘫痪，称为“重症肌无力危象”。发病诱因为精神创伤、过度疲劳、受寒、妊娠、分娩等。多数病人合并胸腺肥大，甚至胸腺瘤，或合并其他自身免疫病。部分患重症肌无力的母亲分娩的婴儿可出现肌无力症状，称新生儿重症肌无力，一般于6周后缓解。诊断可依据以下几点：①疲劳试验阳性。②新斯的明试验阳性。③肌电图神经重频刺激低频波幅递减。④血清AChR抗体滴度升高。治疗手段有：①皮质类固醇。②新斯的明类药物。③胸腺摘除。④胸腺Co60放射治疗。
