细胞核因子κB受体活化因子,Receptor activator of nuclear factor-kappa B英语短句,例句大全

细胞核因子κB受体活化因子,Receptor activator of nuclear factor-kappa B
1)Receptor activator of nuclear factor-kappa B细胞核因子κB受体活化因子
1.Receptor activator of nuclear factor-kappa B/receptor activator of nuclear factor-kappa B ligand/osteoprotegerin(RANK/RANKL/OPG)system plays the key role in Osteoclast\'s activation,development and maturation process.胞核因子κB受体活化因子/细胞核因子κB受体活化因子配体/骨保护因子(RANK/RANKL/OPG)系统在破骨细胞的活化、发育、成熟过程中起关键性作用,近年研究发现该系统也广泛参与骨肿瘤发生、肿瘤骨转移、细胞凋亡,这些研究为相关疾病的治疗提供了新的思路和方法,如重组OPG和人类RANKL单克隆抗体AMG-162的应用。
2)RANKL细胞核因子-κB受体活化因子配体
1.Objective:To study the OPG and RANKL gene expressions in human osteoblast-like MG63 cells seeded on the TNZS disks modified by anodic oxidation.9Sn(TNZS)经过阳极氧化(anodic oxidation,AD)技术处理后,分析其表面的人成骨样MG63细胞骨保护素(osteoprotegerin,OPG)、细胞核因子-κB受体活化因子配体(RANKL)基因表达水平。
3)receptor activator of nuclear factor-κB核因子κB受体活化因子
1.Recombinant soluble receptor activator of nuclear factor-κB inhibits parathyroid hormone-induced osteoclastogenesis in vitro;重组可溶性核因子κB受体活化因子体外抑制甲状旁腺激素诱导的破骨细胞生成

英文短句/例句

1.Changes of receptor activator of nuclear factor kappa B ligand and osteoprotegerin expression in ovariectomized rats去卵巢后大鼠骨组织中核因子κB受体活化因子配体和骨保护素表达的变化
2.Influnce of OPG and RANKL on duck′s osteoclasts formation and activation in vitro骨保护素和核因子κB受体活化因子配基对体外培养鸭破骨细胞的影响
3.Regulation of adult rat condyle receptor activator of nuclear factor kappa B ligand/osteoprotegerin by intermaxillary traction force颌间牵引力对成年大鼠髁状突核因子κB受体活化因子配基/骨保护素的调控
4.A Study on Osteoprotegerin Ligand and Receptor Activator of Nuclear Factor κ-B Express during the Replacement of Dogs Primary and Permanent Teeth;犬乳恒牙替换期骨保护素配体和核因子κB受体活化剂表达的研究
5.Expression of receptor activator of nuclear factor kappa B ligand in root of anterior teeth development in mice核因子κB受体活化剂配体在小鼠前牙根发育中的表达
6.Regulation of receptor activator of nuclear factor-κB ligand-receptor activator of nuclear factor-κB and osteoprotegerin in bone modeling and remodeling核转录因子-κB受体活化因子及其配体和骨保护蛋白系统在骨构建与骨改建中的作用
7.TLR4/NF-κB and Premature BirthToll样受体4/核因子κB与早产
8.Expression of soluble receptor activator of nuclear factor-κB ligand and osteoprotegerin of the os-teoblast in adolescent idiopathic scoliosis patients青少年特发性脊柱侧凸患者成骨细胞中核因子κB受体活化子配体和骨保护蛋白的表达
9.Regulatory Effects of Tea Polyphenols on Peroxisome Proliferator-Activated Receptor α and Nuclear Factor-κB in Alcoholic Liver Disease Models茶多酚对酒精性肝病过氧化物酶体增殖物激活受体α及核因子-κB的调节作用
10.Effect of adiponectin on the expression of osteoprotegerin and receptor activator of nuclear factor-kappa B ligand in osteoblasts脂联素对成骨细胞护骨素和核转录因子κB受体活化素配体表达的影响
11.Osteoclast Differentiation Regulated by Receptor Activator of Nuclear Factor κB Probably Through A Novel Signaling Pathway核转录因子κB受体激活剂可能通过新的信号途径调节破骨细胞分化
12.OSTEOPROTEGRIN AND RECEPTOR ACTIVATOR OF NUCLEAR FACTOR KAPPA B LIGAND mRNAs EXPRESSION IN BMSCs OF GLUCOCORTICOID-INDUCED NECROSIS OF THE FEMORAL HEAD PATIENTS激素性股骨头坏死BMSCs骨保护素/NF-κB受体活化因子配基mRNA的表达
13.HBx Protein Induces Expression of MIG and Increases Migration of Leukocytes Through Activation of NF-κBHBx通过核转录因子NF-κB激活趋化因子MIG表达的机制研究
14.Activity of nuclear factor-κB and cytokines expression in tissues of heart failure rats心力衰竭大鼠组织中核因子κB的活化与细胞因子的表达
15.Nuclear Factor-κB Activation in Peripheral Blood Mononuclear Cells during Acute Kawasaki Disease急性川崎病外周血单个核细胞核因子-κB活化的变化
16.The activation and significance of NF-kappaB in peripheral blood mononuclear cells isolated from subjects with various glucose toleranc不同糖耐量者外周血单核细胞中核因子-κB的活化及意义
17.The Role of Nuclear Factor-kappa B Activation and Fibroblast Phenotype Transdifferentiation in Pulmonary Fibrosis;核因子κB活化与成纤维细胞表型转化在肺间质纤维化中的作用
18.The Regulation of Nuclear Factor-κB Activity by Peptide-Conjugted Decoy ODNs多肽介导的诱骗子对核因子-κB活性的抑制作用

相关短句/例句

RANKL细胞核因子-κB受体活化因子配体
1.Objective:To study the OPG and RANKL gene expressions in human osteoblast-like MG63 cells seeded on the TNZS disks modified by anodic oxidation.9Sn(TNZS)经过阳极氧化(anodic oxidation,AD)技术处理后,分析其表面的人成骨样MG63细胞骨保护素(osteoprotegerin,OPG)、细胞核因子-κB受体活化因子配体(RANKL)基因表达水平。
3)receptor activator of nuclear factor-κB核因子κB受体活化因子
1.Recombinant soluble receptor activator of nuclear factor-κB inhibits parathyroid hormone-induced osteoclastogenesis in vitro;重组可溶性核因子κB受体活化因子体外抑制甲状旁腺激素诱导的破骨细胞生成
4)receptor activator nuclear factor-κB核因子-κB受体活化因子
1.Osteoprotegerin and receptor activator nuclear factor-κB ligand and receptor activator nuclear factor-κB system and tooth eruption;骨保护蛋白-核因子-κB受体活化因子配体-核因子-κB受体活化因子系统与牙萌出
5)receptor activator of nuclear factor-κB核因子κB受体活化剂
1.Osteoprotegerin ligand exerts its biologic effects such as stimulating osteoclasts differentiation, fusion and activating the mature ones through binding to receptor activator of nuclear factor-κB.核因子κB受体活化剂 ,是肿瘤坏死因子受体超家族成员 ,是骨保护素配体在破骨细胞上的受体 ,骨保护素配体通过与核因子κB受体活化剂结合 ,将细胞分化等信号传入破骨细胞前体内 ,促进其分化、融合 ,并促进成熟破骨细胞的激活过程。
2.Objective The purpose of this experiment is to observe the expression of receptor activator of nuclear factor-κB(RANK) and study it s function during the stages of the replacement of dog s primary and permanent teeth.目的　观察犬乳恒牙替换中核因子κB受体活化剂 (receptoractivatorofnuclearfactor -κB ,RANK )的表达 ,探讨其在此过程中所起的作用。
6)RANKL核因子-κB受体活化因子配体
1.Objective To study the effects of tumor necrosis factor-alpha(TNF-α)on the gene expression of receptor activator of nuclear factor-κB ligand(RANKL) and osteoprotegerin(OPG) in human periodontal ligament fibroblasts(HPDLFs).目的探讨肿瘤坏死因子-α(TNF-α)对人牙周膜成纤维细胞(HPDLFs)的核因子-κB受体活化因子配体(RANKL)及其饵受体骨保护素(OPG)mRNA表达的影响,了解TNF-α在破骨细胞性骨吸收调节中的作用机制。
2.The objective of this study was to explore the effect of sodium fluoride on the expression of RANKL to OPG mRNA in mouse osteoblasts.为探讨氟化钠对小鼠成骨细胞中核因子-κB受体活化因子配体(RANKL)和骨保护素(OPG)mRNA表达比率的影响,选取新生小鼠颅盖骨,用胰蛋白酶-胶原酶消化法分离培养成骨细胞。
3.In recent years,the OPG/RANKL/RANK system has made a great breakthrough in bone research field,the results showed that many hormones and cytokines can directly or indirectly regulate the expression of osteoprotegerin(OPG) and receptor activator of NF-κB ligand(RANKL),and control the rate among OPG/RANKL/RANK,thus it could play a role in prevent or promote osteoporosis development.OPG/RANKL/RANK系统是近年来骨科研究领域中的重大突破,研究发现许多激素、细胞因子等均通过直接或间接的调节骨保护素(osteoprotegerin,OPG),核因子-κB受体活化因子配体(receptor activator ofNF-κB ligand,RANKL)的表达,调控OPG、RANKL和核因子-κB受体活化因子(recep-tor activator of NF-κB,RANK)之间的比例,从而介导破骨细胞的分化和功能而达到抗骨质疏松或致骨质疏松的作用。