食管癌细胞株Eca109,human esophageal squamous cancer cell Eca109 line
1)human esophageal squamous cancer cell Eca109 line食管癌细胞株Eca109
1.Objective: To investigate the effect of black currant extract on human esophageal squamous cancer cell Eca109 line to reveal its probable mechanism.方法:采用四甲基偶氮唑蓝(MTT)法测定不同浓度(10-1、10-2、10-3g/ml)黑加仑水提物作用不同时间(12、24、36h)对人食管癌细胞株Eca109的细胞存活量的影响,以大鼠正常肝细胞(BRL)为正常对照细胞株;用Bradford法测定肿瘤细胞蛋白质合成的变化;采用流式细胞技术(FCM)观察黑加仑作用后肿瘤细胞周期变化及凋亡情况;通过倒置显微镜观察细胞的形态变化。
英文短句/例句

1.Effect of oxymatrine on the apoptosis of human esophageal carcinoma Eca109 cell line氧化苦参碱诱导人食管癌细胞株Eca109凋亡的实验观察
2.Arsenic Trioxide Combined with Radiotherapy for the Radiosensitization Mechanism of Esophageal Cancer Cell Line-Eca109三氧化二砷与放射线联合对食管癌细胞株-Eca109的增敏机制
3.Inhibition Effects of RNAi on Telomerase Activation in ECA109 Cells;RNAi抑制食管癌细胞系ECA109端粒酶活性的实验研究
4.Application of ~(18)F-FETNIM for hypoxia monitoring in esophageal cancer ECa109 cells line~(18)F-FETNIM在食管癌ECa109细胞乏氧监测中的应用
5.Role of Stress-activated P38 MAP Kinase in Cisplat and DTT Induced Apoptosis of the Esophageal Carcinoma Cell Line Eca109;应激活化的P38MAPK在DTT与顺铂诱导食管癌Eca109细胞凋亡中的作用
6.An Experimental Study of Resveratrol on the Apoptosis in Human Esophageal Cancer Eca109 Cells and the Expression of Apoptosis-related Genes;白藜芦醇诱导人食管Eca109细胞凋亡及其相关基因表达的实验研究
7.Expression of MT-3 mRNA in human esophageal squamous cell carcinoma人食管鳞状细胞癌细胞株MT-3 mRNA的表达
8.Inhibitory Effects of Flavones and Flavonols on Two Human Esophageal Cancer Cells;黄酮和黄酮醇对两株人食管癌细胞的抑制作用
9.Effect of small interference RNA on expression of the Skp2 in Eca-109 cell linesiRNA干扰对Eca-109食管癌细胞株Skp2表达的影响
10.Effect of Selenomethionine on Cyclooxygenase-2 Expression in Esophageal Carcinoma Cells硒蛋氨酸对食管癌细胞株环氧合酶-2表达影响
11.Inhibition of Nucleostemin(NS) Gene Expression by RNA Interference(iRNA) in Esophageal Cancer Cell LineRNA干扰靶向抑制食管癌细胞株Nucleostemin基因表达
12.Investigation of the esophageal cells ultraweak photon emission and delayed luminescence人食管癌细胞株的超微弱发光与延迟发光
13.RNAi inhibits the expression of FRα in esophageal carcinoma cell line EC109 FRαsiRNA on EC109 ProliferationRNAi抑制食管癌EC109细胞株叶酸受体αmRNA表达的实验
14.The Significances of CpG Island Hypermethylation of MT-3 Gene in Esophageal Squamous Cell Carcinoma;食管鳞状细胞癌细胞株MT-3基因CpG岛超甲基化的意义
15.The Proliferation Effect of Sodium Vanadate on Human Esophageal Cancer Cell Line EC109钒化钠对人食管癌细胞系EC109细胞株增殖影响的研究
16.Effection of cell cycle and radiosensitivity of CPT-11 on Esophageal cancer cell line-109CPT-11对食管癌细胞株ECa-109细胞周期及放射敏感性的影响
17.A Study on Radiosensitivity of β-elemene to Squamous Cell Carcinoma of Oesophagus EC109 Cell Line in Vitro;β-榄香烯乳对食管鳞状癌细胞株(EC109)体外放射增敏作用的实验研究
18.The Research on the Expression of HIF-1 α and Its Relationship with Apoptosis and Proliferation in Esophageal Carcinoma EC9706 Cell Line;HIF-1α表达与食管癌细胞株EC9706凋亡和增殖关系的研究
相关短句/例句

human esophageal squamous cell cancer Eca109 line人食管鳞癌细胞株Eca109
1.Objective: To explore the effect of the DNA methyltransferase-5-aza-CdR on human esophageal squamous cell cancer Eca109 line.方法:应用不同浓度(10-7、10-6、10-5、10-4mol/L)的特异性DNA甲基转移酶抑制剂5-aza-CdR,于不同作用时间(24~168h)处理人食管鳞癌细胞株Eca109,采用四甲基偶氮唑蓝(MTT)法测定5-aza-CdR作用下Eca109细胞株的生存率,倒置显微镜观察细胞的形态学变化。
3)esophageal carcinoma cell ECa109人食管癌细胞株ECa109
1.Objective To study the mechanisms and effect of soybean isoflavone on esophageal carcinoma cell ECa109 during the normoxia and hypoxia.目的研究在缺氧条件下大豆异黄酮(soybeanisoflavone)对人食管癌细胞株ECa109生长抑制作用及可能机制。
4)human Eca109 cell line人食管癌Eca109细胞
5)esophageal cancer cell ECA109人食管癌细胞ECA109
1.Objective To study the inhibitory effects and induction on human esophageal cancer cell ECA109 by As_2O_3.目的探讨三氧化二砷(As2O3)对体外培养的人食管癌细胞ECA109生长的抑制和诱导细胞凋亡的作用。
6)Eca109食管癌细胞Eca109
延伸阅读

癌细胞  生物体内某些正常细胞生长失去控制而恶性增殖的细胞。如果不加切除或抑制它们便会恶性生长,夺取人体养分,使人体消瘦衰竭,最终导致死亡。癌细胞有幼嫩、不成熟、易离散等特点,离散后的癌细胞会随淋巴液和血液循环在体内游走,穿入各种组织器官中继续分裂繁殖,形成新的癌肿,此即癌转移。在离体条件下培养,正常细胞生长到相互接触时就停止增殖,而癌细胞却仍不停止细胞分裂,继续生长成堆,形成不规则团块。大量实验证明,癌细胞生长失去控制,与细胞表面化学物质,特别是表面糖蛋白的变化密切相关。   癌细胞研究已成为生物医学上探索癌变机理的肿瘤生成规律的一个活跃领域。对癌细胞产生的原因,目前还没有一致的见解。一些人认为正常细胞转化为癌细胞是由于致癌病毒诱发的。大多数人同意勃伏利1914年提出的许多癌症的起因是体细胞突变的理论。支持这一观点的论据之一是癌症的发病率随年龄而增长,论据之二是已知能诱发基因突变的所有化学诱变剂或物理因素,几乎都是致癌的,如沥青中的一些化学物质经常接触皮肤,能引起皮肤癌;大剂量苯中毒时,能诱发白血病(血癌);吸烟引起肺癌,这和烟叶中的尼古丁有关;经常接触放射性物质或从事放射线工作的人,白血病、骨髓癌的发病率较高。因此,加强环境保护,消除环境污染,及时检查可疑的致癌物质,并加以防除,对放射性工作加强安全保护等,都可以降低癌症发病率。